申请国外博士 个人简历(CV)

第一篇：申请国外博士 个人简历(CV)

XXX

Gender:Male

Date of Birth: 10/20/1987Add: Nanjing University of Information Science & Technology

School of Environmental Science and EngineeringSchool Office

No.219, Ningliu Road, Pukou District, Nanjing, Jiangsu Province, 210044, P.R.ChinaE-mail:

Tel: 86-***EducationSeptember 2008-Present

Nanjing, China 



Research Experience

February 2011-Present

Research Projects:

 The modification of the antifungal Fluconazole;

 Synthesis of carbohydrate compounds containing β-amino alcohol via Henry

Reaction;

 The synthesis and applications of oxazolidinone compounds.Main Responsibility:

 Search literature, optimize experimental methods under the guidance;

 Carry out reactions and purify the products.March-December 2010

1.Research Projects:

 Design and Applications of Functional Macrocyclic Compounds(College

Students' Science and Technology Innovation Training Projects of Jiangsu Province

(No.xxx and No.xxxx))

2.Chinese Patent:

 The Synthesis and Applications of Macrocyclic Compounds Containing The

Structure of Arylamine(Application No.xxxxxxxxx.x).3.Publication

 Synthesis and characterization of the novel shape-persistent nanosized cavity

macrocycle,synthetic metals 2011, XXX, XXXX-XXXX.Research Interests

 Synthetic organic chemistry in natural products;

 Asymmetric synthesis.English Proficiency

 IELTS: 6.5(Listening 6.5, Reading 7.5, Writing 6, Speaking 6.5)

Professional Skills

Chemistry

 Proficient in literature review, spectrum analysis and normal laboratory techniques; Enable to conduct reactions with air sensitive reagents using a double manifold; Monitor reactions with TLC, HPLC, GC;

 Purify and separate products using column chromatography.Computer

 Specialized software: ChemBio Office, Origin, Endnote, etc.;

 Literature searching online.Extracurricular Activities and Awards











President Scholarship, NUIST, Oct.2011 Award of Pacemaker to Merit Student, NUIST, Oct.2011 Award of Excellent Student Cadre, NUIST, Oct.2011 Pei Zhi Scholarship, NUIST, Jan.2011 First Prize in the 2nd Chemical Experiment & Skills Contest of NUIST, Jan.2011

Hobbies

 Badminton, Jogging, Reading, Music

第二篇：CV英文个人简历

personal Information:

Name: XXX

Sex: Male

Date of Birth: XX XXth XX

place of birth: XX, p.R.China

Marital Status: XX Health: Good

Mobile :13XXXXXXX

E-mail: wdjlw@www.xiexiebang.com

Objective:

Application Engineer, Technical and Electronics Sales.Education Background:

2002.9??2006.6 XXXX.Bachelor of Electronic informationn engineering

Major subjects: Circuit principle, Single Chip Computer principle and Application, Digital Electronic Technology, Analog Electronic Technology, Interfacing and Application of Computer, principle of Communication,Work Experiences:

2006.7-present XXX As a computer software engineer, Responsibilites includes :

1.Solving environmental problems on prototype computers.Conducte systems analysis on new computer profucts to ensure hardware,software and mechanical design integrity

2.And responsible for System compatibility and integration test.Quickly diagnose causes of systems failures and malfunctions to ensure highest operating efficiencies,reliability,and quality performance standards.3.Assist engineers from Hp wIth installation and trobleshooting of Hp Bussiness pC software

English Skills

1.pass CET-4

2.Have a good command of spoken and written English.Communicate and negociate with

clients fluently in English.3.Skilled in reading documents relating to Electronics and Computer

Others

Independent and be able to work under pressue.Have coordination skills,teamwork spirit.Studious

nature and dedication are my greatest strengths

第三篇：(CV)英文个人简历正规格式

DENG XIAO PENGMobile: XXXXXX;Email: XXXXXXX

Name:

Gender: male

E-mail:

Tel:

Experience

2011-2012English intern in a poor mountain village elementary

school.2012-2013college student union.Manage the things that used in some big event.Education

2012.9-presentstudy in Wenzhou University, major in Business English.Main Courses American CultureHistory and Anthology of English LiteratureMicro-economyReading Course in American and British News Publications English-Chinese Translation The Art of Public Speaking A Survey: Major English-Speaking Countries Oral English Computer Skills

Strength

 English language skill: fluent in using English for work and communications

 Fast adaptation to new working environment and dedicated to efficient team

work







Strong experience in team building, project management and coordination.Project planning, budget control and manage deliverables on schedule.Business development and customer oriented communication.Interests

Playing music, badminton, chatting with friends, reading, English films,writing.

第四篇：出国留学申请CV

留学申请CV模板与范文

今天讲CV先，之前收到小叽同学的CV，打开一看，GPA旁边还粘了一张用EXCEL做的GPA四年曲线表，把我昏过去了…所以决心先写CV篇。CV，两个字加三个符号：Be Professional！!一定要看上去专业，CV是你的门面，你的脸，所有的申请，读书也好实习也好工作也好征婚也好，了解一个人，就凭这张纸，把自己化妆得漂亮点，关键是要让人信得过，让人觉得你靠谱，让人觉得你professional！

我三年前申请的时候，也没有任何人教，到处查CV的模板，自己反复改了很多遍改到烦死了，最后就递交了算了，时间也来不及。

后来到了这里，学校有专门的就业指导中心，帮你改CV，各个学院也专门有针对各个学院的，我跑过很多个，也认真研习了各种学校提供的CV参考模板，有很系统的一套。我写这个系列的目的就是指出CV写作的要点，剖析常见错误。我以前犯过的，你们不要重蹈覆辙。

我每年都更新我的CV，主要是确实有需要的场合用到，第一年07年申请学校，第二年08年申请夏天的实习，第三年09年申请工作，后来又申请GA，针对每个工作内容都改一版新的。“针对”很重要！内容要针对！语言和排版要专业！就这两个。

我为了写这个CV篇重新翻出我当年申请时候的CV，和之后每年更改的CV，现在看当年的，简直是相形见绌，哭笑不得，那幼稚的，拿不出手，遮脸遮脸。那么Pirate Radio（非常好看英伦摇滚电影 海盗电台）里面说，能承认错误的，才称得上是个男人。

话说，CV和RESUME，的区别，是RESUME就一页，你人生之精华，就缩在一页之内，是给人家一瞟眼了解你用的。

而CV，一个好习惯的且professional的人，是该每隔半年就更新一遍的，是你的编年史，每发生了一点变动，有新的内容，就补充上去，是长长的，细细的，是为了已经对你感兴趣的人想更深入的了解你用的。比如RESUME里你一句话“2007年担任iCow社团主席”

而CV里在这句话下面加五个标记点： “2007年担任iCow社团主席：

-建立了全国第一家大学生有机奶牛农场；出席了2007国家牛奶生产加工会议；为社区以及大学学生生产了13加仑的有机牛奶；

参与拟定了第17项国家奶牛保护法”

我上面这样写不够对仗，正式的简历，如果想好了以动词开头，每一个句子的第一个词都要动词开头，如果是副词“独立担当了”，下一个就要以“团队形式地合作了”，要词性对仗，懂？ 所以我以上应该改成： “ iCow社团主席 2007生产了13加仑的有机牛奶为社区以及大学学生；参与拟定了第17项国家奶牛保护法”

我把第二句的顺序移动了，这样人家 一瞟眼 RESUME的一扫，第一竖列，都是动词，都是行为，句子都是动宾结构，这样让人家很容易看懂，你颠来倒去，人家只要有一个wonder，你到底在写什 么，马上心里的好感会降下来，马上会引起心烦，那你present你的RESUME是要设想，是一个给教授 按摩的过程，把人家弄舒服，人家才愿意招你一起合作。

精细到如此精心的准备你的RESUME，才算合格。

BTW，以动宾开头最好，action 的动词很catch人家的眼球。

6.每一件事情发生的时间要标出来。简历上精确到季度或年份就可以了。美国这边对时间很注意，随便什么地方签名旁边都要标个时间，支票也是，发票也是，不要说一般的文件表格了。7.GPA尽量算成4分制。

美国按照4分制的, 国内有些学校按照5分的，换算成4分制，否则来个4.XX的GPA这里还要给你重新算，那你还不如最开始就用最能把自己的GPA算高的算法算出来呈现给对方呢

8.用逗号分隔句子，而不是用分号.分号在英语里的用法我到现在都吃不准，和中文里的分号用法是不一样的。所以如果要分隔句子，用逗号。

Conducted research on…., managed team project xxx, and developed xxxx 而不是

Conducted reseach on…;managed team project xxx;developed xxxx 9.给没有研究背景的非牛牛们再来一道法子: Course project

我在这里第一个学期读研究生的时候，才读了几个月，实习的招聘会就开始了。我都还没学什么呢，更表说研究背景了。我去就业中心问这个简历要怎么写 写，我刚来，都没这个专业的背景。对方说，不要慌，大部分学生都没研究背景的，把你学的课程列出来，你的课业里的作业列出来，就把那些作业作为你的 project experience。

美国这里的课程作业，做项目的多，有的是团队项目，有的是个人的，一般研究生都是独立的项目。比如我做过一个要去学生宿舍楼里检测安装设备的，还有 一个是大楼的险情状况计算机模拟，一个项目一般给一个月的时间，从文献综述，编写程序或者出访调查，到写份几十页的报告，到PPT演讲。这一类的作业很练 人。做完了就真的懂很多。就业中心的指导说，把这类作业写到你的简历里去，比如你的一门作业，老师布置，每个学生采集一头奶牛的奶水样本，你在简历上就 写：

-独立采集大学有机奶牛农场牛奶样本 写得好听一点呢，可以说：

-在大学有机奶牛农场做牛奶生产检测

很多学工科的学生，大多有些实验课，把这些课的内容写出来：-研究了17种传感器的工作原理，包括…… 社会学科和商科大多有调查课题：

-调查了17头奶牛哺乳期的心理活动，对此撰写了一份17页的英文调查报告，并正式演讲了调查发现(formally presented the results / findings / it)就业指导说，大家都是学生，就是没有什么特别尖锐的学术研究背景的，就把课堂里的作业写出来，真的一个一个去想去列做过哪些作业，认真的漂亮的措词写到CV上，就会变成一份很丰富的简历，人家一看你做过很多事情。简历好不好看的关键在于你如何present你的过去。

本来么，你确实做过很多事情，你确实在某门课的作业里设计了某个电路，确实写了十篇实验报告，为什么不摆出来说呢？

不要以为只有研究生的或者跟着导师发论文的研究才算得上研究背景，你读了4年大学，每个人的轨迹不一样，别人花很多时间在实验室里，你必定把这些时 间花在了别的事情上，除了逛街买衣服磨脸打游戏，你比别人多做的有什么，如何安排你的时间，4年时间的一个专业的学习，肯定是做过不少跟专业相关的事情 的。

对方的招生或者教授，看的也就是你： a.有没有做研究的能力 b.有没有写论文的的能力 c.有没有做演讲的能力

你如果在一门实验课里做了10个实验，那就是有很好的动手能力，把它拿出来说呀，这些经历无非不是在某个特别的导师的办公室里做的而已，就是课堂做 的，怎样，我要体现的就是，我懂电路，我懂编程，我懂模拟，我懂传感，我懂数学模型……你要体现的不就是你有这个能力么？纠结于它是在哪里完成的干嘛？

我以前也笨的，明明课堂作业里做过很多小测试小project，但总觉得那是作业啊，那不算什么啊，那人人都做的作业啊，不是高深的研究啊。所以当时一个都没放，只觉得专业背景空空。

其实是错的，明明做过很多跟专业相关的事情，你跟导师研究室里做出来的东西，无非只是课堂实验室的延长版，一样的本质啊。

所以，每个人都必定有起码5行的专业研究背景可以写，请打印出你们的成绩单一门一门课去想，在那门课里做过了什么吧。

我当初到了这里第一学期才读了几个月的课，一共三门课，要我写简历，我就把三门课里做的project一个一个列写出来，一门课里有三个project，另一门课里有7个实验，另一门课里有一份大报告，就这样整整列了一整页的简历，简历就满满的很有内容很漂亮了。

他招生单位怎么知道你在学校每门课的老师都布置什么作业？他招的来自这么多学校的怎么会知道你们学校什么课程设置和要求？ 他要看到的就是你有经验，有能力。你要呈现的也就是“我能”。简历的关键在于如何呈现。

不要说你想想自己一片空白，过去没做什么。

给我认真冥想，仔细列出来。丰满你的简历吧 10.用数字说话。

小叽给我的简历里有一项她组织文艺活动的内容，写 “Organized many musical parties”

我读下去，知道她写的是她组织过很多活动

首先“parties”这个词在美国多指疯狂小本们的周末酒精派对，用词不准，用events更合适

再，many 这个词不好，没有力道，这个时候就要用数字，改成

“Organized more than twenty campus-wide events including two annual chorus contests, three New Year Gala, …., etc.” 11.想用引号的时候，用斜体而不是引号。

这里也有个标点问题。中文里用引号的地方，英文里往往是用斜体。比如： 我的研究课题是“天花板下的母奶牛” 在英文简历里改成

我的研究课题是 天花板下的母奶牛

研究课题啦，三好学生 之类的奖学金称号啦，书名啦，最好都改成斜体，而不是引号，比较符合英语习惯。我到现在都不知道双引号在英文里怎么用的，很少看到。

简历中表示强调没有什么死规定，粗体，斜体，下划线，自己安排。保持前后一致，不要太疯狂就好。

没有错，双引号和斜体应当都可以。

而且在参考文献里，对期刊，会议论文等引用格式不同，有一种用的是双引号没错。

但我的日常接触中，除了引用别人说话的内容用双引号，其他地方很少见。尤其是在非参考文献的场合，比如一本书里作者提及另一本书的标题，在我以往接触到的资料里，往往常见的是斜体，而非引号。

简历中提及自己毕业设计的名字，或者课程项目的名字，不是正规的已经发表了的论文，我认为 斜体 较 引号 更为妥当。

第五篇：申请国外大学博士的计划书

Research Interest Proposal I am the Doctor degree applicant Xu Zhiwei.From September 2008 to June 2013, I studied the laboratory medicine at Nantong University.In September 2013 I was recommended to the graduate school by the professors due to my excellent assessment.Through undergraduate studies, I contacted such theories as molecular biology, cellular biology , immunology and so on.However, keenly conscious that my current acquisition is far from enough for me to meet the needs of the fast developing medicine, I am eager to pursue further studies in Chinese medical sciences.With my hard working, I know about the basic knowledge of medical immunology.Up to now, I have known a lot about the University of Macau.The graduate school of university plays a prominent role in the academic research and transforms Macao into a knowledge-based society.I am dedicated to pursuiting medical research and hoping to come to the university of Macau for rich experiences and success in PhD program.I am now applying for Chinese Medical Science in the University.This planned research has the following aims: The deubiquitinating enzyme USP14 has been identified and biochemically studied, but its mechanisms in cancer remains to be elucidated.Protein glycosylation with O-linked N-acetylglucosamine(O-GlcNAc)is a reversible post-translational modification occurring onserine or threonine residues.Intriguingly, it has been observedthat O-GlcNAcylation is particularly abundant on cancer cells.The aim of this study was to evaluate the O-GlcNAcylation of USP14 in patients with cancer and to define its mechanisms in cancer cell proliferation and apoptosis.a.To demonstrate that O-GlcNAcylation of USP14.b.To explore Interplay between USP14 O-GlcNAcylation and phosphorylation c.To determine

how

O-GlcNAcylation

regulates

metabolic reprogrammingand Signaling in cancer cells.d.To test whether O-GlcNAcylation regulates proliferation and apoptosis.2.Research Context

Deregulating cellular energetics is emerging as a characteristichallmark of cancer cells.Withinsuch cells, glucose and glutamine are used at an increasedrate, resulting in the production ofATP in a manner independent of oxygen concentration.Elevated glucose and glutamine flux areneeded not only to serve the energetic demands of cancer cells,but also to provide the essential carbon and nitrogen used inmacromolecule synthesis, fueling the rapid growth and proliferation seen in tumors.This increasein glucose and glutamine uptake can alter multiple metabolicand signaling pathways in cancer cells, including for examplethe hexosamine biosynthetic pathway(HBP).The HBP relies on glucose and glutamine uptake, and approximately 3%–5% of the total glucose entering a cell is shunted intothis pathway.This critical metabolite isrequired for the biosynthesis of a variety of extracellular glycopolymers, including both N-and O-glycans, however, it also serves as the substrate for O-linked b-N-acetlyglucosamine

(O-GlcNAc)

transferase

(OGT).O-GlcNAcylation is directly involved in growth hormone(gibberellic acid)signalling in plants, and both SPY and SECRET AGENT(SEC)encode O-GlcNAc transferases.Mutations in either SPY or SECcause severe growth defects;simultaneous mutation of both genes islethal.Unlike plants, mammals and insects seem to have only a single gene encoding the catalytic subunit of the O-GlcNAc transferase(OGT)Gene disruption in

mice

established

that

OGT

is

required forembryonic-stem-cell viability20.Tissue-targeted disruption in miceshowed that O-GlcNAcylation is essential to several cell types.OGTdeletion causes hyperphosphorylation, which is followed by cell death, induces T-cell apoptosis and causes growth arrest infibro blasts.Cre–lox-mediated deletion of OGT in cultured fibroblastsresults in death as pre-existing OGT protein levels diminish.This modification can be removed by theglycoside hydrolase O-GlcNAcase(OGA)that catalyzes cleavage of O-GlcNAc from proteins.This modification can alter protein functiondirectly or, in some cases, by competing with phosphorylationsites.O-GlcNAc and O-phosphate site-mapping studies suggest that there areat least four different types of dynamic interplay between O-GlcNAcand O-phosphate.First, there is competitive occupancy at thesame site, for example that which occurs in the transcription factorc-Myc25 and oestrogen receptor-β26, and on the oncoprotein SV-40 largeT-antigen27 and endothelial nitric oxide synthase28.Second, competitiveand alternative occupancy occur at adjacent sites, such as that observedin the tumour suppressor p53 and synapsin I.Third,there is a complex interplay whereby some O-phosphate attachmentsites on a given protein are the same as some O-GlcNAc sites, whereasothers are adjacent to, or even distant from, each other, such as on theC-terminal domain of RNA polymerase II and on cytokeratins32.The final type of interplay involves proteins in which this relationship has yet to be clearly defined.The interplay between O-GlcNAcand O-phosphate is also underscored by the recent finding that OGTtransiently forms complexes containing the catalytic subunit of proteinphosphatase 1(PP1c).Cancer cells, however, uptake glucose at a higher rateand produce lactic acid rather than metabolizing pyruvate throughthe tricarboxylic acid(TCA)cycle.This adaptive metabolic shift is termed the Warburg effect, leading to anaerobic glycolysis, and is thought toprovide an evolutionary advantage to cancer cells by providingboth increase bioenergetics and biosynthesis.Many protooncogenes(e.g., Ras and Myc)and tumor suppressors(e.g., p53)influence metabolism,and mutations in these genes can upregulateglucose uptake in cancer cells and promote a metabolic phenotype supporting

tumor

cell

growth

and

proliferation.Elevatedglucose uptake in cancer cells can be applied to monitor the location of primary and metastatic tumor sites;for an example, usingF-18 fluorodeoxyglucose(FDG), a glucose analog, with a combination of positron emission tomography/computed tomography(PET/CT).Recent study has providedinsights into the mechanism ofpost-translational modification of molecules in cancer cells theregulation of many molecules and

suggested

important

implications

in

cancer development.Additionally, lines ofevidence of global proteomic analysis havesuggested that post-translational modifications of USP14 are likely not limited to phosphorylation.Other forms of modifications, such asO-GlcNAcylationappear to occur as well,suggesting a more sophisticated regulatorynetwork of USP14.In this study, we would evidence that O-GlcNAcylation within cancer cells regulates cancer cell metabolism via regulation of phosphorylation and its downstream target genes.Mechanistically, we wonder which signaling pathway has participated in the regulation.Furthermore, we will discuss whether decreased O-GlcNAcylation leads to reduced proliferation and apoptosis incancer cells.In addition, we hypothesized that human cancers containing high USP14 levels also containelevated OGT and O-GlcNAcylation.Importantly, we will explore in overallcancer patients, lower OGA expression correlates withpoor clinical outcome.Thus,we will confirm that O-GlcNAcylation serves as a criticallink between the key pathways thatare critical for cancer cell survival via regulation of glycosylation.Method: a.To demonstrate that O-GlcNAcylation of USP14.Here, the O-GlcNAc moietyon the protein is labelled with UDP-GalNAz using a mutantgalactosyltransferase GalT1 Y289L(mGalT1)with an azidederivative of UDP-GalNAc(UDP-GalNAz)as donor substrate,followed by labelling with biotin alkyne.After in vitro O-GlcNAcylation, USP14 was subjectedto mGalT1 labelling and then detected by probing withstreptavidin-conjugated HRP.b.To explore Interplay between USP14 O-GlcNAcylation and phosphorylation.Wewill explore that whether increasing USP14 O-GlcNAc modification with GlcN orPUGNAc treatment inhibits NF-κB activation and have delineatedthe molecular mechanisms of this effect.We will demonstrate that USP14 is a target for O-GlcNAc modification inGlcN or PUGNAc treated cells, and that this post translationalmodification prevents its phosphorylation in response to TNF-α,suggesting a reciprocal relationship between O-GlcNAcylation andphosphorylation of USP14 in cancer cells.We would further showthat, in cancer cells pretreated with GlcN or PUGNAc, levels of O-GlcNAcylation and phosphorylation of USP14 was changed.c.To determine

how

O-GlcNAcylation

regulates

metabolic reprogrammingand Signaling in cancer cells.Since OGT and O-GlcNAc has been associated with regulationof metabolic diseases such as insulin resistance, we hypothesized that OGT could serve as an importantregulator of glycolytic metabolism to regulate cancer cell growth.To test this idea, we initially examined the effect of OGT reduction on metabolites from human cancercells using liquid chromatography-mass spectrometry(LC-MS).The metabolic profile of cancer cellscontaining OGT knockdown with RNAi demonstrated a generaldecrease in glycolytic and pentose phosphate pathway(PPP)metabolites and an increase in tricarboxylic acid(TCA)cycle metabolites, consistent with a reversalof the Warburg effect and inhibition of cancer cell growth underthese conditions that we and othershave previously shown.d.To test whether O-GlcNAcylation regulates proliferation and apoptosis.Glucose deprivation and antiglycolytic drugs can selectivelyinduce tumor cell proliferation and death;thus, we examined the effect of reducing O-GlcNAcylation on proliferation and apoptosis in nontransformed immortalized mammary epithelial cells compared tocancer cells.In cancer cells stablyexpressing control siRNA or OGT siRNA at day 8 post-infection,we we tested whether cell rounding and detachment of cancer cells containing OGT knockdown, while cancer cells attached to the plate.To further investigate the effect of OGT SiRNA on cellular proliferation, we used chemically synthesized siRNA to knockdown endogenous OGT in cancer cells.The efficiency of the OGT-targeted siRNA-mediateddown-regulation was assessed by Western blot analysis.Aspredicted, siRNA knocked down the protein expression of OGT as compared with negative control siRNA and mocktreatment.To determine the effect of OGT knockout on cancer cell proliferation, OGT-siRNA, negative control-siRNA and mock treatment cancer cell proteins were testedby Western blot.We would explore that expressed decreased OGT levels hadelevated cleaved caspase-3 and caspase-8, and upto 50%–70% of cells were examined for annexin V.